Skip to main content
PharmSpace

Class Explorer · Anti-infectives

Macrolides

Bind the 50S ribosomal subunit and block translocation. Bacteriostatic, excellent intracellular and atypical coverage, and the interaction profile of a CYP3A4 inhibitor.

Members

Also in this class: Erythromycin. Full pages arrive as the library grows.

Compare the members

Azithromycin
CYP3A4
Minimal inhibition
Half-life
Very long, tissue
Niche
Short courses, fewer interactions
Clarithromycin
CYP3A4
Potent inhibitor
Half-life
3 to 7 h
Niche
H. pylori regimens

Shared across the class

Indications
Community-acquired pneumonia, atypical cover · Pertussis · Chlamydia · H. pylori eradication (clarithromycin)
Adverse effects
Gastrointestinal upset · QT prolongation · Hepatotoxicity, uncommon
Contraindications
Known QT prolongation with other risk factors · Concurrent drugs with dangerous CYP3A4 dependence
Monitoring
QT where other QT-prolonging drugs are in play · INR if the patient is on warfarin · Clinical response

What makes each agent different

  • Clarithromycin is a potent CYP3A4 inhibitor and the source of the statin, calcium-channel-blocker and warfarin interactions this class is known for. Azithromycin barely touches CYP3A4, which is its main practical advantage.
  • Azithromycin's tissue half-life is measured in days, which is what allows three- and five-day courses that keep working after the last tablet.

The macrolide plus statin combination is a rhabdomyolysis question every time: with clarithromycin the usual answer is to hold the statin for the course.

Both raise cardiovascular risk slightly through QT effects; in a patient already collecting QT-prolonging drugs, doxycycline is often the quieter choice.

Last reviewed 2026-08-16 · Goodman & Gilman's The Pharmacological Basis of Therapeutics, 14th ed.