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Systemic corticosteroids · Endocrine

Prednisone

prednisolone (active form) · Deltasone® · Winpred®(brand names vary by market)

The most useful drug on the ward, and the one that quietly does the most damage.

Illustration of Prednisone as a tabletPRE

Also: oral liquid

Mechanism

A prodrug converted to prednisolone, which binds the glucocorticoid receptor and alters transcription of hundreds of genes: less cytokine production, less leukocyte trafficking, and effects on glucose, bone, mood and the adrenal axis that come with it.

Indications

  • Asthma and COPD exacerbations
  • Autoimmune and inflammatory disease
  • Giant cell arteritis and polymyalgia rheumatica
  • Transplant and haematological protocols
  • Adrenal insufficiency, in combination

Formulations

TabletOral liquid

Dosing concepts

Short course

Courses of under about three weeks can usually be stopped abruptly, because the adrenal axis has not had time to suppress.

Long course

Beyond roughly three weeks, or with repeated courses, the axis is suppressed and the drug is tapered. The taper is not about the disease, it is about the adrenal gland.

Timing

Given in the morning to mirror the natural cortisol rhythm and reduce insomnia.

Sick days

A patient on long-term steroids who becomes ill needs MORE, not less; that is the opposite of the instinct and it is life-saving.

Concepts, not prescribing instructions. Practice numbers live in current references and local protocols.

Pharmacokinetics

Bioavailability
~80%
Half-life
Plasma 2 to 4 h; biological effect 12 to 36 h
Elimination
hepatic: Converted to prednisolone by the liver, so prednisolone is used directly in significant liver disease
Protein binding
~70 to 90%
Play with these concepts in the PK Lab

Adverse effects

  • Hyperglycemiacommon & seriousCharacteristically afternoon and evening, so a fasting morning glucose can miss it entirely.
  • Insomnia, mood change, agitation, frank psychosiscommonDose-related and often in the first week.
  • Increased infection riskserious
  • Osteoporosis and fractureserious
  • Adrenal suppressionserious
  • Weight gain, Cushingoid appearance, skin thinning, bruisingcommon
  • Peptic ulceration, mainly alongside NSAIDsserious
  • Cataract and glaucoma with prolonged useserious

Contraindications & precautions

Contraindications

  • Systemic fungal infection
  • Live vaccines at immunosuppressive doses

Precautions

  • Diabetes
  • Peptic ulcer disease or concurrent NSAIDs
  • Psychiatric history
  • Osteoporosis
  • Latent tuberculosis or hepatitis B

Interactions

Monitoring

Glucose

Steroid hyperglycemia is near-universal at higher doses and often missed · From the first days in anyone diabetic or at risk, using afternoon readings

Blood pressure and weight

Fluid retention and metabolic effects

Bone protection assessment

Anything beyond about three months warrants calcium, vitamin D and a bisphosphonate discussion

Mood and sleep

Common, distressing, and rarely asked about

Counselling points

  • Take it in the morning, with food.
  • Do not stop it suddenly if you have been on it for more than a few weeks; the dose is reduced gradually.
  • Carry a steroid card, and tell any clinician treating you that you take it.
  • If you become unwell, contact us: the dose usually needs to go up, not down.
  • You may feel unusually awake, irritable or hungry. Tell us if your mood changes markedly.
  • Report any sign of infection promptly, and stay away from chickenpox and measles if you have not had them.

Clinical pearls

  • Steroid-induced hyperglycemia peaks in the afternoon and evening. Checking only a fasting glucose is the standard way to miss it, and insulin regimens for it are shaped accordingly.
  • The single most dangerous moment in long-term steroid therapy is an intercurrent illness during which the patient reduces the dose because they feel unwell.
  • Prednisone requires hepatic conversion to prednisolone; in significant liver disease, prednisolone is prescribed directly.

References & review

  • Prednisone product monograph (consult the current version for your market) (monograph)
  • Goodman & Gilman's The Pharmacological Basis of Therapeutics, 14th ed. (reference-work)

Last reviewed 2026-08-16 · jurisdiction: global · drug information changes; verify against current references before practice use.